Heat-stressed cells use nuclear stress bodies to restart RNA splicing, study finds
If you want to beat the heat of the summer sun, slowing down and doing less is a good strategy.
If you want to beat the heat of the summer sun, slowing down and doing less is a good strategy.
The short version
- However, researchers have long asked whether the same occurs at the cellular level.
- While cellular stress responses have been repeatedly studied, the impact of the environment on these responses remains relatively unknown.
- This article has been reviewed according to Science X's editorial process and policies .
What happened
Editors have highlighted the following attributes while ensuring the content's credibility: If you want to beat the heat of the summer sun, slowing down and doing less is a good strategy. Now, researchers from Japan report an elegant mechanism by which cells shut down certain nonessential functions when they get too hot.
Why it matters
In a study recently published in Molecular Cell , researchers from the University of Osaka revealed how nuclear stress bodies sense temperature to regulate survival during and recovery from thermal stress.
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